glutathione deficiency amino acids Metabolism - an overview Glutathione deficiency and heart failure:

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However, analyzing a dsbB mutant strain regarding its oxidation state and re-oxidation capacity in presence or absence of GSH or GSSG revealed that GSSG was still able to complement for the loss of DsbB, indicating a DsbB-independent mechanism (Supplementary figure 4)

The peak selection coefficients were the VIP (variable importance of projection) from the OPLS-DA model

The p53 (3KR) mutantin which three lysine residues are substituted with arginines, abolishing acetylationspecifically reduces SLC7A11 expression without altering other p53 targets such as CDKN1A/p21 (a cell-cycle regulator) or BAX (an apoptosis-related protein)