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Excessive DA signaling may regulate gene expression and modify the synaptic function and circuit activity (68)

In vitro studies show that GHK-CU boosts endothelial cell proliferation, migration, and tube formation

Furthermore, the accumulation of neutral lipids resulting from the deletion of adipose triglyceride lipase (ATGL) in the endothelium leads to impaired endothelial-dependent vascular tone and reduced nitric oxide synthesis, exacerbating endothelial dysfunction

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