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IFN-I signaling promotes Chac2-mediated GSH reprogramming and N1-type polarization of neutrophils To identify upstream regulatory pathways responsible for the observed GSH metabolic reprogramming in BM neutrophils during periodontitis, we performed gene set enrichment analysis (GSEA) of neutrophil transcriptomes from Lig and Con mice

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It reduces the iron concentration by converting the peroxidised lipids accumulated during ferroptosis into non-toxic isoalcohols (36,37)