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Recent studies of OA cartilage homeostasis have focused on the increase in senescent cells in articular cartilage [4,5,6] can result from mitochondrial dysfunction, excessive mechanical loading, oxidative stress, and DNA damage [7,8,9,10]

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Mutation of clk-1 and isp-1 [42], RNAi of cyc-1 [43], and RNAi of spg-7 [39] have all also been shown to activate mitochondrial UPR

Smoking and environment-related lung injury Smoking is the most common environmental factor contributing to lung injury, and the respiratory tract is also chronically exposed to particulates, dust, smoke, and other inhaled toxins