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propose visual cycle modifications as a treatment option (ALK001-P3001, NCT03845582), via the use of a modified form of vitamin A that replaces natural vitamin A in the body, thus slowing the production of damaging vitamin A dimers, postulated to slow the accumulation of toxic end products and therefore slow the progression of AMD (2019d)

Therefore, even in the presence of higher PGC-1 gene expression as a result of mTOR hyperactivity during early AMD stages, a reduction in PGC-1 activity in later stages could ultimately affect mitochondrial turnover and biogenesis causing increased ROS production and cell death

Because the skin is the protective barrier to the environment, it can receive direct oxidative damage in the epidermis

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