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Taken together, these findings demonstrate that while GSSG is indeed formed during plasma treatment, extensive treatment can cause an over-oxidation of the disulphide bond resulting in the two end products GSNO and GSO 3 H with a strong preference for the latter

18 When coupled with smaller and more infrequent meals, bile salt secretion into the intestine is decreased, reducing gallbladder motility and triggering the formation of biliary sludge, i.e., crystals or microliths composed of precipitated lipid particles

(2010, October 3)

is inhibited by high extracellular Glu concentration, and decreasing intracellular GSH leads to peroxidase 4 (GPX4) inhibition and lipoxygenase (LOX) activation