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Adequate folic acid intakes can normalize high homocysteine levels via increased remethylation of homocysteine to methionine via 5-methyltetrahydrofolate-homocysteine methyltransferase (a.k.a

This finding indicated that the Trp prototrophy inhibited V-ATPase-dependent autophagy via Trp and/or its derivative metabolites

Testing for MMA and homocysteine levels is considered a more accurate reflection of metabolic B12 activity (or B12 thats available for your body to actually use)

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