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Metabolic advantages of regulatory T cells dictated by cancer cells [J]

Shapiro LLM, Whitehead SA, Thomas MB

A key future direction will be the rational design and testing of dosing regimens that explicitly account for the formation, maintenance, and exit of drug-tolerant cancer cells, which may enable more mechanistic interpretation of clinical outcomes and improve the translational relevance of intermittent or adaptive treatment strategies

Downregulating the expression of COPZ1 in U-87MG and U251 cells induces ferritin autophagy and increases intracellular iron levels, eventually leading to ferroptosis [97]