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Figure 4 Alterations in the intracellular redox status are often associated with GSH depletion (Polonikov, 2020) and endogenous GSH deficiency, due either to decreased biosynthesis and/or increased consumption, is a marked contributor to the pathogenesis of numerous diseases via mechanisms including oxidative stress and inflammation

RSL3 treatment significantly reduced chondrocyte ATP generation compared to the control group, but pioglitazone treatment boosted cellular ATP production compared to the RSL3 group (Fig

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