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The conserved mechanisms of the SSP, including redox homeostasis, one-carbon metabolism, and drug resistance regulation, can serve as common targets for cross-cancer therapeutic interventions

This mechanism involved changes in hepatocyte-macrophage crosstalk and LXR activation, which we identified to be critical for pro-resolving macrophage phenotype and the fibrosis resolution process

Burton EM, Voyer J, Gewurz BE (2022) Epstein-Barr virus latency programs dynamically sensitize B cells to ferroptosis

and the effectiveness of VEST in preventing vein graft disease 12 months post-CABG was better in all subgroups, compared to the control group