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This enhances the activity of the promoter and thus IGF-1R acts as a transcriptional auto-activator [3]

Liu C, Yan X, Zhang Y, et al

Lahiri DK, Maloney B, Greig NH

Current challenges include the absence of specific biomarkers and targeted inhibitors, necessitating preclinical validation of disulfidptosis-inducing agents to advance precision oncology strategies for SLC7A11-driven malignancies (Figure 7) (Table 3)