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Dewanjee S, Chakraborty P, Bhattacharya H, Chacko L, Singh B, Chaudhary A, et al

In research models, this makes it useful for examining how reduced NNMT activity changes intracellular nicotinamide handling, methyl donor utilization, and downstream metabolic signaling

In keeping with the reduced capacity of homocysteine-exposed endothelial cells to inhibit platelet aggregation, Lentz and collaborators demonstrated, using experimental hyperhomocysteinemia (induced by a diet depleted of folate, high in methionine, and choline-free) that the endothelial-dependent vasodilation is impaired in hyperhomocysteinemic primates

doi: 10.1111/J.1365-3040.1995.TB00615.X 69 GargN.ManchandaG