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It has been demonstrated that the missense variant NQO1, found in 4-20% of the human population, results in no detectable NQO1 activity due to rapid degradation of the NQO1 P187S protein

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Takahashi et al., 2006) and have been detected in atherosclerotic plaques, heart valves, aortic aneurysms, carotid arteries, and coronary vessels (Marques, da Silva et al., 2005

Furthermore, compared to normal controls, individuals with eating disorders have also shown decreased GSH synthesis, and a therapeutic dose of APAP could potentially be an overdose, complicating the livers ability to reduce the toxic metabolite NAPQI in the case of APAP-induced hepatotoxicity [20]