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Glutathione metabolism in the lung: Inhibition of its synthesis leads to lamellar body and mitochondrial defects

The primary mode of action for these bacteriocins involves either pore formation in the target cell membrane or inhibition of cell wall synthesis, ultimately resulting in bacterial cell death (196, 197)

Inhibition of non-CYP enzymes especially UDP-glucuronosyltransferases (UGTs) significantly predicts high risk for drug-induced liver injury (DILI)

In both normolipidemic C57/Bl6 and aged atherosclerotic ApoE-deficient mice, GA improved vascular cell adhesion molecule function and inhibit platelet activation prior to platelet activation