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The transporter SLC38A1 and the amino acid transporter SLC1A5-mediated glutamine uptake and subsequent glutaminase 2 (GLS2)-mediated glutamate production are required for cysteine-deprivation-induced ferroptosis [43, 44]

Remarkably, ~5090% reduction of L68Q-hCC staining was observed in five of the treated carriers suggesting that L68Q-hCC is a clinical target for reducing agents

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Though this is perhaps one of its most prominent and perhaps obvious functions, research in recent years has shed light on the potential other functions of glutathione that are perhaps equally as important in cell maintenance and regulating controlled cell death, with implications for tendency to develop cancer