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By inhibiting initiation and propagation steps, they terminate reactions, delay oxidation, and counteract oxidative mechanisms in disease progression 177,178

Activation of ADRB2 counteracted the behavioral and synaptic deficits in FTO-deficient mice, with the ADRB2 agonist formoterol alleviating depression-like behavior and restoring dendritic spine density, while an antagonist blocked these effects

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PCSK6 was found up-regulated in CRC, and recent studies have shown that PCSK6 participates in proliferation and migration of tumor cells in different human malignancies, but its regulatory mechanism in CRC remains unclear [14]