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Mitochondria-to-nucleus signaling promoted stress-responsive and proinflammatory transcriptional programs, while mitochondrial failure in the apical secretory region indirectly facilitated defective exocytosis and premature zymogen activation

The therapeutic efficacies of this IC were similar to those of interferon- treatment and the protocol for treatment was deemed to merit further optimization 83 Importantly, transient flares of alanine aminotransferase (ALT), reflecting injuries in liver function, were observed in a small percentages of IC-treated patients, but liver functions became normal during treatment or at the follow-up period

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Structure of GPX4 U46C-R152R is modeled based on PDB structure 2OBI