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Feng and colleagues [90] reported that acrolein exposure to human lung cells produces a pattern of DNA damage in the p53 tumor suppressor gene which appeared to be similar to the p53 pattern of mutations found in lung cancer

Newer combinations of drugs and multimodal treatment regimensimplemented in both neoadjuvant and adjuvant settingshave shown promise in extending survival

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Mice fed the KD had higher mitochondrial respiration rates and increased expression of mitochondrial uncoupling proteins (UCP) than mice on a control diet (57)