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This inhibition primarily results from the suppression of both cell elongation and division at the root apex 8,9,10

Various general autoantibodies have also been identified in ME/CFS, such as those against cellular components including anchorage molecules (514), HSP-60 (515), microtubule associated protein 2 (516), cardiolipin in 92-95% of ME/CFS patients in two studies (509, 510) but only 4% in another study (506), and neo-antigens (517)

But the most noteworthy immune processes occur in the synovial membrane and synovial fluid (Nygaard and Firestein, 2020

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