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This antibody, when interacting with PD-1, reinstates cytokine release from activated T cells, effectively blocking both the HGF/c-MET and PD-1/PD-L1 signaling pathways [144]

For example, peak levels of ovarian hormones during proestrus is correlated with increased levels of proliferation in the SGZ [138, 139]

For the self-resistance of c-Met inhibitors, the combination of c-Met inhibitors and other inhibitors can be used to solve this problem, such as FGFR and EGFT inhibitors, autophagy inhibitor and anti-PD1 treatment

Data availability The original contributions presented in the study are included in the article/supplementary material