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Introduction The Developmental Origins of Health and Disease (DOHaD) hypothesis, first articulated by Barker and colleagues in the late 20th century, provides a conceptual framework for understanding how environmental exposures during critical periods of development shape long-term health trajectories [1,2]

The presence of no-reflow at TP2 was associated with worse functional outcomes (mRS 0-1: OR 0.14, 95%CI 0.020.84, p = 0.03) but not at TP1 (OR 0.16, 95%CI 0.021.33, p = 0.09)

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BPC-157 addresses CAI through multiple mechanisms