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For instance, we have identified a confirmed target pathway for anti-fungal drugs with glutathione metabolism (GSH), attributed to fungal mitochondrial maintenance, preservation of membrane integrity, regulation of transcription factors in stress response and protection against reactive oxygen species

Current research suggests anodal tDCS induces a subthreshold, polarity-dependent membrane polarization that induces neural plasticity via N-methyl-D-aspartate, gamma-aminobutyric acid, brain-derived neurotrophic factor, and calcium-dependent mechanisms [76, 274,275,276]