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These interactions could inadvertently alter the metabolism, systemic exposure, and toxicity profiles of co-administered chemotherapeutic agents, underscoring the need for careful pharmacological monitoring in combination regimens

In line with the importance of TXNRD1 in the regulation of ferroptosis and apoptosis in AML cells, auranofin, a TXNRD1 inhibitor, impaired cell viability in AML subtypes dependent on TXRND1 for their protection against ferroptosis [53]

We suggest that the NO and SNO-dependent abnormal alterations of the key biochemical and biological processes in the brain (discussed above) led to the behavioral deficits of SA-treated mice

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