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It was also reported in DRG neurons of wild type and TRPV4 knockout mice that activation of TRPV4 by paclitaxel induced mechanical allodynia and excessive ROS production, although the allodynia and oxidative stress was partially decreased by the TRPV4 antagonist treatment (Materazzi et al., 1)

Selenocysteine-mediated native chemical ligation

Hypothalamicpituitaryovarian axis and control of the menstrual cycle

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