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In vitro experiments have demonstrated that uric acid can cause vascular endothelial cell injury by activating the NLRP3/IL-1 signaling pathway or Nicotinamide adenine dinucleotide phosphate (NADPH) enzymes (17)

dynamic processes such as changing organ perfusion, metabolism, distribution into peripheral compartments, and bedside dose titrationknown to shape catecholamine pharmacokinetics in vivoare not reproduced in this closed-loop model 4,5

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Analyzing the molecular pathomechanism of axon fraction, including intra-axonal transcription factor, could provide a clue to elucidate the fragility of motor neurons in ALS