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Mahapatra, S., et al

The homeoprotein Nanog is required for maintenance of pluripotency in mouse epiblast and ES cells

Normally, at an optimal dose, acetaminophen is effectively metabolized by CYP2E1 and CYP1A2 enzymes to form a toxic metabolite, NAPQI, which is known to play a major role in drive oxidative stress-induced hepatoxicity through rapid conjugation with GSH [71]

In mouse models, the team introduced combined therapy including PD-L1 blocker and cyst(e) inase (a bioengineered enzyme that degrades cystine and cysteine) and such treatment can enhance the efficacy of CTL-mediated anticancer effects, supporting the idea that inducing ferroptosis could be considered a productive way for treating cancer and overcoming immunotherapy resistance (Wang et al., 2019)