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Key factors underpinning neuroimmune-metabolic-oxidative (NIMETOX) major depression in outpatients: paraoxonase 1 activity, reverse cholesterol transport, increased atherogenicity, protein oxidation, and differently expressed cytokine networks

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Hypoxia-reprogramed megamitochondrion contacts and engulfs lysosome to mediate mitochondrial self-digestion

This suggests that BPC-157 could hold promise for conditions such as traumatic brain injury, stroke, and neurodegenerative diseases, where preserving neuronal function and promoting recovery are critical therapeutic goals