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Treatment with NR improves cognition in transgenic mice with AD by reducing the phosphorylation of the Tau protein (pTau) (a form that accumulates in the brain of AD patients and is the hallmark of the disease) (Green et al., 2008

At least three cytoprotective systems against ferroptosis with distinct subcellular localizations have been identified in recent studies: glutathione peroxidase 4 (GPX4) located in the cytoplasm and mitochondria

The reviewed studies demonstrate that ASD-like behavioral deficits in preclinical models can be modulated through interventions targeting diverse biological systems, including neurotransmission, neuroinflammation, metabolism, and the gut-brain axis

Some research suggested potential adaptogenic properties the ability to help organisms maintain homeostasis when faced with physical, chemical, or biological stressors