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Mechanically, artesunate upregulated the expression of autophagy-associated proteins and inhibited the expression of FTH1 and NCOA4, indicating that ferritinophagy-mediated ferroptosis in HSCs is crucial in the antifibrotic therapy of artesunate (Kong et al., 2019)

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Research shows that low B12 levels associate with higher testosterone deficiency risk and worse androgenic hormone profiles that affect sperm production [5]