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Geissmann, T.A., Teuber, M., and Meile, L., J

Curtis, Photographer by Ralph W

When acutely administered in the perfusate at non-necrotic concentrations, tBOOH impaired bile salt excretion and increased the bile/perfusate ratio of [14C]- sucrose, a tight-junctional-integrity marker that enters the bile canaliculus via the paracellular pathway.70 The impairment in bile salt excretion and the disruption of the tight-junctional barrier function may be causally associated, since the latter induces leakage of bile salts (or other osmotically-active biliary solutes) from the canalicular space via the paracellular pathway, leading to dissipation of the osmotic gradients involved in bile formation.112 These functional alterations to secrete and retain bile salts in the biliary space seem to have a structural correlate