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Apart from directly stimulating hepatocyte proliferation, KCs also engage with other subsets of non-parenchymal cells, indirectly influencing and enhancing the liver regeneration process through cellcell interactions or paracrine signaling via the production of inflammatory factors

While our study design and statistical methods for association testing account for population structure to mitigate confounding, these inherent EHR biases may impact the overall power and generalizability of the findings across different ancestral groups

Genetic Factors Leading to Deficiency Genetic mutations can result in severe forms of glutathione reductase deficiency, with deficient subjects displaying electrophoretic polymorphisms linked to the deficiency

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