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Mutation-independent therapies can have various downstream targets that aim to improve mitochondrial respiration, reduce mitochondrial stress, inhibit or delay RGC apoptosis, and/or promote RGC survival

doi: 10.3390/ijms22052734 32 YanHWangLZhangGLiNZhaoYLiuJet al

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Docking accelerates the identification of potential therapeutics by modeling intermolecular interactions and optimizing complementarity and structural precision