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Consistent with their higher FDFT1 expression, released cells showed greater resilience to functional inhibition, highlighting FDFT1-driven metabolic priming as a mechanism supporting robust proliferation after transient cell cycle blockade

Mice expressing the human APOE4 allele exhibited reduced metastasis relative to APOE2 mice, suggesting certain genetic lesions can increase metastatic competence

Thiosulfate was also produced in a similar manner (Supplementary Figs S2 and S4d)

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