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Furthermore, mutations in several genes were enriched in post-T-DXd samples compared to unmatched T-DXd-nave samples, including ERBB2 , NFE2L2 (a transcriptional activator of ABCC1 ), and KEAP1 (a negative regulator of NFE2L2), indicating plausible resistance mechanisms related to HER2 target levels and ABCC1-mediated drug efflux

L., Emerling, B

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