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While CNS drugs like sertraline (SRT) have shown promise in animal models, where their combination with front-line anti-TB drugs, improved early bactericidal activity, resolution of pulmonary pathology, and enhanced survival in mice infected with M.tb [70], clinical studies have raised concerns

CM-specific overexpression of placental growth factor (PlGF) stimulates myocardial angiogenesis and increases endothelial release of NO, resulting in myocardial hypertrophy via degradation of RGS4, a regulator of G-protein signaling involved in the CM hypertrophic response ( Animal models have also been critical in confirming mechanistic drivers of CMD in the context of comorbid metabolic derangements (Sorop et al., 2020)

Galactofuranose biosynthesis starts with the isomerization of UDP-galactopyranose to UDP galactofuranose by UDP galactomutase encoded by the glfA gene

Singh B, Chaudhary R, Katharia R, Pradhan M