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( C ) The decreased GSH:GSSG increases LDH release, consistent with activation of oxidatively driven cell death ( n = 6)

We also discuss multiple factors that influence exercises effect and highlight the importance and need for further investigations regarding the exercise-regulated molecules as therapeutic targets and biomarkers for CVD as well as the cross talk between the heart and other tissues or organs during exercise

The special feature noted in the present study could be responsible, in particular, for the increased expression and internalization of VEGFR2, which is known to be essential for endothelial function, and the activation of the VEGFR2-Akt-eNOS signaling pathway[25]