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Materials and methods Reagents Arsenicals, iAs (Sigma S7400, NaAsO 2 , purity equal or more than 90%) prepared as a 10 mM stock in ddH 2 O was further diluted in H 2 O to the desired concentrations

Patients who developed HCC before or within six months of baseline were excluded

Conclusion: Differential expression of gut microbiota could underlie the pathogenesis of lean NAFLD, although it is difficult to label it as an entirely distinct pathophysiological entity

Their well-defined porous structures facilitate O 2 diffusion into the material and efficient release of ROS outward, thereby enhancing ROS generation during PDT and SDT (144150)