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EAE, induced by the peptide myelin oligodendrocyte glycoprotein (MOG), served as a clinically relevant model to study the dynamics of vascular lesions and the associated molecular alterations

Tyrosine phosphorylated occludin could delocalize/disassemble from the tight junctions and undergo proteasome-mediated degradation, a phenomenon observed in patients suffering from inflammatory bowel syndrome 79,80

Am Psychol

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