lippard glutathione cisplatin in cancer therapy: molecular mechanisms of action Molecular mechanisms of cisplatin resistance

(2.1, 4.2, or 8.4 g/kg) for six weeks alleviated hepatic steatosis, oxidative stress, and inflammation in ALD rats via the gutliver axis by modulating the overall structure of the GM, such as maintaining the Firmicutes/Bacteroidetes ratio and reducing the relative abundance of harmful bacteria including Clostridium and Faecalibaculum, while activating the 15d-prostaglandin J2/Peroxisome proliferator-activated receptor (PPAR) and PPAR signaling pathways to lower serum levels of TNF-, IL-1, and IL-6, thereby improving lipid metabolism [121]

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Acta 1794 , 254262 (2009)

Septic cardiomyopathy (SIC) In a model of LPS-mediated septic cardiomyopathy, some researchers revealed that LPS caused ferroptosis and septic cardiac injury by downregulating GPX4 protein levels and increasing COX-2 [93]