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PP6-driven loss of T-loop phosphorylation could lower binding affinity between TPX2 and Aurora-A and ultimately cause release of the kinase into the cytoplasm, making it available for centrosomal recruitment by CEP192

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A recent study showed that Trx1 attenuates cigarette smoke extract-induced endoplasmic reticulum (ER) stress, ROS production, mitochondrial dysfunction, and mitophagy-mediated inflammasome activation while enhancing the activation of the Nrf2/HO-1 signaling pathway, contributing to the improved antioxidant and anti-inflammatory response in murine lungs [130]