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Description

Int J Cancer (2009) 124:170715

Impairment in the insulin signaling reduces the affinity of insulin for the insulin receptor When insulin signaling is impaired, there is an alteration in the interaction between insulin receptors with insulin receptor substrate 1(IRS1) and insulin receptor substrate 2 (IRS2)

While neutrophil-derived reactive oxygen species (ROS) play an important role in breaking down damaged fragments of the muscle tissue, ROS produced in excess may also contribute to oxidative stress (for review see [47, 48]

However, with the recognition of apoptosis and its detailed signaling mechanisms, reports were published suggesting that APAP also triggers apoptotic cell death [40, 41]