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In the face of controversies and challenges, it is essential to establish a more efficient translational interface between science, technology, and clinical practice, driving mitochondrial metabolic reprogramming therapy from a potential pathway to clinical reality. Conclusion Mitochondrial metabolic reprogramming plays a central role in the therapeutic failure of CRC

Two to six times more ynfA gene expression than tolC gene expression was observed (Sarkar et al., 2015)

This allowed for multivalent binding to the EGFR receptor, overexpressed on inflammatory breast cancer (IBC) cells, and ultimately enabled the delivery of the enzyme intracellularly

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