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Results Molecular analysis reveals structural stability of GRLX and pKa of individual cysteines Given the previous observations that GLRX can be inactivated via an oxidative mechanism 8,23 , herein we sought to investigate the relative vulnerability of each of the individual cysteines towards GSSG, a model glutathionylating agent, or H 2 O 2 and their contribution to oxidative inactivation

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Moreover, the release of IL-18 at 10 M doses and IL-8 at 10 M and 20 M doses were significantly increased in contrast to LVX (Fig

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