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SARS-CoV-2 gains cellular entry via its receptors angiotensin-converting enzyme 2 and transmembrane protease serine 2, but it may enter via other receptors, including neuropilin and vimentin, all of which are enriched in cells of the neurovascular unit 6,7,8,9,10

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[9] For wound healing specifically, the evidence is stronger than for cosmetic anti-aging, because wound models involve measurable endpoints (wound closure rate, tissue tensile strength, histological analysis) in controlled animal studies

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