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Description

Grx2 knockout or overexpression exacerbates or diminishes lipid peroxidation in human lens epithelial cells or opacity in mouse lenses, respectively, after treatment with ferroptosis inducers or UV-B via modulation of mitochondrial structure and the GSH/GSSG ratio [128]

It has been reported that WNT5A served as a key regulatory of endothelial inflammation and could promote the expression of tissue factors and platelet-activating factors, which would further affect DVT progression [51, 52]