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These results suggest that the cytotoxicity observed following treatment with MitoQ alone and in combination with DPI is not solely dependent on its ability to regulate mitochondrial superoxide production in MNA NB cells, which is in line with recent studies on MitoQ effects in other cancer cell types (31, 32)

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In pre-clinical trials (primarily rodent models), SLU-PP-332 has demonstrated an exceptional safety profile

This occurs because the MTHFR enzyme plays a crucial role in the conversion of homocysteine to methionine