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Notably, these two different genomic compositions of enhanced interaction areas are connected by the key antioxidant enzyme superoxide dismutase 2 (SOD2), showing potential functional synergy

inhibiting GPX4 is also a promising strategy to overcome tumor cell resistance to conventional chemotherapy drugs by triggering ferroptosis as an alternative cell death mechanism [75]

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Plasma levels of trimethylamine-N-oxide are confounded by impaired kidney function and poor metabolic control