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This is especially interesting for GBM patients, since galectin-1 has been identified as a poor prognostic factor, fulfilling a key role in the aggressiveness and immune escape of GBM cells [68, 72, 102,103,104]

Either the GHK-CU isn't suitable for your biochemistry, your protocol needs adjustment, or an external factor is interfering

For example, the level of -glucuronidase in gut metagenomes was higher in males than in females, providing a potential explanation for sex-based differences in efficacy and toxicity for several drugs [118]

ROS in TAO are produced by infiltrating immune cells, orbital fibroblasts and adipocytes, leading to tissue damage and inflammation