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[PubMed] Brustolin, S., Giugliani, R., & Flix, T

Brown, T., & Lee, Y

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For example, endothelium-derived NO directly suppresses the electrical excitability of VSMCs by inhibiting the action of T-type and L-type voltage-gated Ca 2+ channels in small arteries, and reduced NO availability can trigger transient depolarization in normally quiescent VSMCs leading to vasospasm (Smith et al., 2020)