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Retrieved January 12, 2020

PF prevented the upregulation of pro-inflammatory mediators (TNF-, IL-1b, iNOS, COX2, and 5-LOX) in plasma and brain to the sustained activation of hippocampal AST and microglia caused by chronic cerebral insufficiency of cerebral perfusion or cerebral ischemia, suggesting that PF exerts delayed protective effects in ischemia-injured rats by inhibiting the peripheral and cerebral tissue inflammatory responses mediated in MAPKs/NF-kB (Guo et al., 2012

It is legally available only for scientific studies and research purposes in the United States

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